Varicella-Zoster virus Retains study Histocompatibility Complex Class I Proteins in the Golgi Compartment of septic Cells Allison Abendroth, Ines Lin, Barry Slobedman, Hidde Ploegh and Ann M. Arvin J. Virol. 2001, 75(10):4878. inside: 10.1128/JVI.75.10.4878-4888.2001. These include: REFERENCES CONTENT ALERTS This article cites 47 articles, 24 of which can be accessed free at: http://jvi.asm.org/ character/75/10/4878#ref-list-1 Receive: RSS Feeds, eTOCs, free email alerts (when innovative articles cite this article), more» Information about commercial reprinting orders: http://journals.asm.org/ station/misc/reprints.xhtml To subscribe to to another ASM Journal go to: http://journals.asm.org/ rouse/subscriptions/ Downloaded from http://jvi.asm.org/ on August 21, 2012 by guest Updated information and go can be found at: http://jvi.asm.org/content/75/10/4878 JOURNAL OF VIROLOGY, May 2001, p. 48784888 0022-538X/01/$04.00 0 DOI: 10.1128/JVI.75.1 0.48784888.2001 copyright © 2001, American Society for Microbiology. All Rights Reserved. Vol. 75, No.
10 Varicella-Zoster Virus Retains Major Histocompatibility Complex Class I Proteins in the Golgi Compartment of septic Cells ALLISON ABENDROTH,1 INES LIN,2 BARRY SLOBEDMAN,1 HIDDE PLOEGH,3 AND ANN M. ARVIN2* nerve for Virus Research, Westmead Millennium Institute of Health Research, University of Sydney, Sydney, virgin South Wales, Australia1; Departments of pediatrics and Microbiology & Immunology, Stanford University School of Medicine, Stanford, California2; and Department of Pathology, Harvard Medical School, Boston, Massachuset! ts3 true 14 July 2000/recognised 15 February 2001 Varicella-zoster virus (VZV) is a piece herpes virusvirus that causes varicella (chicken pox) as the primary infection in susceptible individuals, establishes latency in sensory nerve ganglia, and may reactivate as herpes zoster (shingles) (5, 6, 14). In wakeless individuals, primary VZV infection induces...If you want to present a respectable essay, order it on our website: OrderCustomPaper.com
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